🧪
hypothesis

Domain boundary cross-talk hypothesis

Hypothesis

Domain boundary cross-talk hypothesis

MAP6 stability activity may suppress adjacent tau lability activity through direct physical interaction or by altering tubulin post-translational modifications.
🧬 MAP6🩺 neurodegeneration🎯 Composite 60%💱 $0.54▼1.2%proposed
EvidenceModerate (55%)📖 6 cit🗣 1 debates 6 support 2 oppose
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Mechanistic 0.65 (15%) Evidence 0.60 (15%) Novelty 0.60 (12%) Feasibility 0.60 (12%) Impact 0.00 (12%) Druggability 0.00 (10%) Safety 0.00 (8%) Competition 0.00 (6%) Data Avail. 0.00 (5%) Reproducible 0.76 (5%) KG Connect 0.30 (8%) 0.600 composite
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🧪 Overview

MAP6 stability activity may suppress adjacent tau lability activity through direct physical interaction or by altering tubulin post-translational modifications

Prediction: MAP6 overexpression will extend stability into adjacent tau-rich regions; MAP6 knockout will extend lability into MAP6-rich regions

🧬 Mechanism

🧬 Curated Mechanism Pathway

Curated pathway from expert analysis

flowchart TD
    A["MAP6 Occupancy on Microtubules<br/>Cold-Stable Cytoskeletal Support"]
    B["Tau/MAPT Lattice Competition<br/>Dynamic Binding Balance"]
    C["Synaptic Remodeling Signals<br/>NMDA-Linked Cytoskeletal Plasticity"]
    D["Axonal Transport and Branching<br/>Circuit Adaptation"]
    E["MAP6-Tau Imbalance<br/>Rigid or Unstable Cytoskeleton"]
    A --> B
    B --> C
    C --> D
    E -.->|"disrupts"| B
    style A fill:#1a237e,stroke:#4fc3f7,color:#4fc3f7
    style D fill:#1b5e20,stroke:#81c784,color:#81c784
    style E fill:#b71c1c,stroke:#ef9a9a,color:#ef9a9a

⚖️ Evidence

⚖️ Evidence Matrix6 supports2 contradicts
Supports
MAP6 stability activity may suppress adjacent tau lability activity through direct physical interaction or by altering tubulin post-translational modifications
Supports
Tau and MAP6 establish labile and stable domains on microtubules.
iScience2025PMID:40040809medium
Supports
Antagonistic roles of tau and MAP6 in regulating neuronal development.
J Cell Sci2024PMID:39257379medium
Supports
BioID2-Based Tau Interactome Reveals Novel and Known Protein Interactions Associated with Multiple Cellular Pathways.
J Proteome Res2025PMID:40910579medium
Supports
Tau: It's Not What You Think.
Trends Cell Biol2019PMID:30929793medium
Supports
Beyond Neuronal Microtubule Stabilization: MAP6 and CRMPS, Two Converging Stories.
Front Mol Neurosci2021PMID:34025352medium
Contradicts
Tau/MAP6 antagonism is shown in neuronal development models, but this does not establish that the same balance drives adult neurodegeneration progression or treatment response.
J Cell Sci2024PMID:39257379medium
Contradicts
A microtubule-stabilizing peptide strategy failed to show clinical benefit in progressive supranuclear palsy, cautioning against simple cytoskeletal-stabilization translation in tauopathy.
Lancet Neurol2014PMID:24873720medium
📖 Linked Papers

No linked papers recorded for this hypothesis yet.

🏥 Translation

🧬 3D Protein Structure — MAP6

No curated PDB or AlphaFold mapping for MAP6 yet. Search RCSB →

🧠 GTEx v10 Brain ExpressionJSON

Median TPM across 13 brain regions for MAP6 from GTEx v10.

Cortex42.4 Frontal Cortex BA941.4 Cerebellum36.7 Hypothalamus35.9 Nucleus accumbens basal ganglia34.4 Cerebellar Hemisphere34.0 Anterior cingulate cortex BA2433.1 Caudate basal ganglia27.9 Spinal cord cervical c-125.5 Amygdala24.3 Hippocampus24.1 Putamen basal ganglia22.3 Substantia nigra18.0median TPM (GTEx v10)

💉 Clinical Trials (1)Relevance: 70%

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No curated ClinVar variants loaded for this hypothesis.

Run scripts/backfill_clinvar_variants.py to fetch P/LP/VUS variants.

🔍 Search ClinVar for MAP6 →

No DepMap CRISPR Chronos data found for MAP6.

Run python3 scripts/backfill_hypothesis_depmap.py to populate.

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🏆 Arenas / Elo

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💾 Resource Usage

No resource usage or linked notebooks recorded for this hypothesis yet.

🔮 Predictions

🔎 Predictions vs Observations2 predictions · 0 with recorded observations
PredictionPredictedObservedStatusConf
IF MAP6 is genetically ablated in iPSC-derived cortical neurons using CRISPR/Cas9, THEN the proportion of neurons exhibiting cytosolic mislocalization of tau (co-localization ratio <0.3 with axonal maAt least 40% of MAP6-knockout neurons showing tau mislocalization vs. <20% in controls, quantified by high-content immunofluorescence imaging and automated neur— no observation —pending0.38
IF MAP6 is overexpressed 2-fold in primary mouse cortical neurons via AAV2/9 transduction, THEN the ratio of pS396-tau to total tau in detergent-soluble fractions will decrease by at least 30% compareDecreased tau phosphorylation at Ser396 by ≥30% in MAP6-overexpressing neurons as measured by quantitative western blot (normalized to β3-tubulin loading contro— no observation —pending0.45
🔮 Falsifiable Predictions (2)
pendingconf 45%
IF MAP6 is overexpressed 2-fold in primary mouse cortical neurons via AAV2/9 transduction, THEN the ratio of pS396-tau to total tau in detergent-soluble fractions will decrease by at least 30% compared to control-transduced neurons within 7 days post-transduction
Predicted outcome: Decreased tau phosphorylation at Ser396 by ≥30% in MAP6-overexpressing neurons as measured by quantitative western blot (normalized to β3-tubulin load
Falsification: No significant change (<15%) in pS396-tau/total tau ratio between MAP6-overexpression and control conditions, or increased rather than decreased phosphorylation
pendingconf 38%
IF MAP6 is genetically ablated in iPSC-derived cortical neurons using CRISPR/Cas9, THEN the proportion of neurons exhibiting cytosolic mislocalization of tau (co-localization ratio <0.3 with axonal marker Tau1) will increase by at least 2-fold compared to isogenic control lines within 21 days of dif
Predicted outcome: At least 40% of MAP6-knockout neurons showing tau mislocalization vs. <20% in controls, quantified by high-content immunofluorescence imaging and auto
Falsification: No difference (<1.3-fold) in tau mislocalization between MAP6-KO and control lines, or tau correctly localized in >70% of MAP6-KO neurons
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