🧪
hypothesis

Activity-dependent MAP6 scaffolding at synapses

Hypothesis

Activity-dependent MAP6 scaffolding at synapses

MAP6 may scaffold signaling complexes at synapses in an activity-dependent manner, linking NMDA receptor activation to cytoskeletal remodeling via its multiple functional domains.
🧬 MAP6🩺 neurodegeneration🎯 Composite 60%💱 $0.53▼1.5%proposed
EvidenceModerate (55%)📖 6 cit🗣 1 debates 6 support 2 oppose
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Mechanistic 0.65 (15%) Evidence 0.60 (15%) Novelty 0.60 (12%) Feasibility 0.60 (12%) Impact 0.00 (12%) Druggability 0.00 (10%) Safety 0.00 (8%) Competition 0.00 (6%) Data Avail. 0.00 (5%) Reproducible 0.76 (5%) KG Connect 0.30 (8%) 0.600 composite
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🧪 Overview

MAP6 may scaffold signaling complexes at synapses in an activity-dependent manner, linking NMDA receptor activation to cytoskeletal remodeling via its multiple functional domains

Prediction: MAP6 will physically associate with synaptic signaling proteins in a phosphorylation-dependent manner, and LTP-inducing stimulation will recruit MAP6 to dendritic spines

🧬 Mechanism

🧬 Curated Mechanism Pathway

Curated pathway from expert analysis

flowchart TD
    A["MAP6 Occupancy on Microtubules<br/>Cold-Stable Cytoskeletal Support"]
    B["Tau/MAPT Lattice Competition<br/>Dynamic Binding Balance"]
    C["Synaptic Remodeling Signals<br/>NMDA-Linked Cytoskeletal Plasticity"]
    D["Axonal Transport and Branching<br/>Circuit Adaptation"]
    E["MAP6-Tau Imbalance<br/>Rigid or Unstable Cytoskeleton"]
    A --> B
    B --> C
    C --> D
    E -.->|"disrupts"| B
    style A fill:#1a237e,stroke:#4fc3f7,color:#4fc3f7
    style D fill:#1b5e20,stroke:#81c784,color:#81c784
    style E fill:#b71c1c,stroke:#ef9a9a,color:#ef9a9a

⚖️ Evidence

⚖️ Evidence Matrix6 supports2 contradicts
Supports
MAP6 may scaffold signaling complexes at synapses in an activity-dependent manner, linking NMDA receptor activation to cytoskeletal remodeling via its multiple functional domains
Supports
Cerebellin-neurexin complexes instructing synapse properties.
Curr Opin Neurobiol2023PMID:37209532medium
Supports
Sensory neurons drive pancreatic cancer progression through glutamatergic neuron-cancer pseudo-synapses.
Cancer Cell2025PMID:41005304medium
Supports
mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists.
Science2010PMID:20724638medium
Supports
Distinct neurexin-cerebellin complexes control AMPA- and NMDA-receptor responses in a circuit-dependent manner.
Elife2022PMID:36205393medium
Supports
Beyond NMDA Receptors: Homeostasis at the Glutamate Tripartite Synapse and Its Contributions to Cognitive Dysfunction in Schizophrenia.
Int J Mol Sci2022PMID:35955750medium
Contradicts
Tau/MAP6 antagonism is shown in neuronal development models, but this does not establish that the same balance drives adult neurodegeneration progression or treatment response.
J Cell Sci2024PMID:39257379medium
Contradicts
A microtubule-stabilizing peptide strategy failed to show clinical benefit in progressive supranuclear palsy, cautioning against simple cytoskeletal-stabilization translation in tauopathy.
Lancet Neurol2014PMID:24873720medium
📖 Linked Papers

No linked papers recorded for this hypothesis yet.

🏥 Translation

🧬 3D Protein Structure — MAP6

No curated PDB or AlphaFold mapping for MAP6 yet. Search RCSB →

🧠 GTEx v10 Brain ExpressionJSON

Median TPM across 13 brain regions for MAP6 from GTEx v10.

Cortex42.4 Frontal Cortex BA941.4 Cerebellum36.7 Hypothalamus35.9 Nucleus accumbens basal ganglia34.4 Cerebellar Hemisphere34.0 Anterior cingulate cortex BA2433.1 Caudate basal ganglia27.9 Spinal cord cervical c-125.5 Amygdala24.3 Hippocampus24.1 Putamen basal ganglia22.3 Substantia nigra18.0median TPM (GTEx v10)

💉 Clinical Trials (1)Relevance: 70%

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No curated ClinVar variants loaded for this hypothesis.

Run scripts/backfill_clinvar_variants.py to fetch P/LP/VUS variants.

🔍 Search ClinVar for MAP6 →

No DepMap CRISPR Chronos data found for MAP6.

Run python3 scripts/backfill_hypothesis_depmap.py to populate.

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💾 Resource Usage

No resource usage or linked notebooks recorded for this hypothesis yet.

🔮 Predictions

🔎 Predictions vs Observations2 predictions · 0 with recorded observations
PredictionPredictedObservedStatusConf
IF neuronal activity is selectively increased via chemogenetic activation of excitatory neurons in cultured hippocampal networks, THEN MAP6 protein will show a significant increase in synaptic spine lIncreased MAP6 accumulation at dendritic spines as measured by live-cell imaging of MAP6-GFP expressing neurons— no observation —pending0.65
IF MAP6 phosphorylation-deficient knockin mice (S→A mutations at predicted activity-regulated sites) are subjected to theta-burst LTP induction, THEN the maintenance phase of LTP (60-120 min post-induReduced LTP maintenance magnitude (≤150% of baseline) at Schaffer collateral-CA1 synapses in knockin vs wild-type mice— no observation —pending0.58
🔮 Falsifiable Predictions (2)
pendingconf 65%
IF neuronal activity is selectively increased via chemogenetic activation of excitatory neurons in cultured hippocampal networks, THEN MAP6 protein will show a significant increase in synaptic spine localization (≥40% increase in spine/cytoplasm ratio) within 60 minutes post-stimulation.
Predicted outcome: Increased MAP6 accumulation at dendritic spines as measured by live-cell imaging of MAP6-GFP expressing neurons
Falsification: MAP6 spine/cytoplasm ratio changes by <20% or shows no change despite robust neuronal activity increase (c-Fos upregulation ≥3-fold confirms activation)
pendingconf 58%
IF MAP6 phosphorylation-deficient knockin mice (S→A mutations at predicted activity-regulated sites) are subjected to theta-burst LTP induction, THEN the maintenance phase of LTP (60-120 min post-induction) will show significantly reduced potentiation compared to wild-type littermates.
Predicted outcome: Reduced LTP maintenance magnitude (≤150% of baseline) at Schaffer collateral-CA1 synapses in knockin vs wild-type mice
Falsification: LTP maintenance in knockin mice remains within 10% of wild-type magnitude (both ≥160% baseline), indicating MAP6 phosphorylation is not required for LTP
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