🧪
hypothesis

G2019S Acts as Lysosomal Volume-Sensing Amplifier via Enhanced RAB29-Dependent Recruitment (H1)

Hypothesis

G2019S Acts as Lysosomal Volume-Sensing Amplifier via Enhanced RAB29-Dependent Recruitment (H1)

G2019S specifically hyperactivates LRRK2 when recruited to swelling lysosomes via RAB29, creating pathogenic positive feedback where membrane stress increases RAB10 phosphorylation more than wild-type.
🧬 LRRK2,RAB29🩺 neurodegeneration🎯 Composite 73%💱 $0.58▼14.7%proposed
EvidencePending (0%)📖 8 cit🗣 1 debates 8 support 2 oppose
✓ All Quality Gates Passed
Mechanistic 0.68 (15%) Evidence 0.72 (15%) Novelty 0.78 (12%) Feasibility 0.82 (12%) Impact 0.85 (12%) Druggability 0.70 (10%) Safety 0.72 (8%) Competition 0.65 (6%) Data Avail. 0.68 (5%) Reproducible 0.70 (5%) KG Connect 0.50 (8%) 0.730 composite
🏆 ChallengeResolve: G2019S LRRK2 Creates Pathogenic Positive Feedback as Lysosomal Volume-S$500K →
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Composite73%

🧪 Overview

G2019S specifically hyperactivates LRRK2 when recruited to swelling lysosomes via RAB29, creating pathogenic positive feedback where membrane stress increases RAB10 phosphorylation more than wild-type. Key experimental prediction: rise kinetics (slope) should differ between G2019S and WT, not merely baseline offset. RAB29 pathogenic mutations (PARK23) confirm disease relevance of this axis.

🧬 Mechanism

🧬 Curated Mechanism Pathway

Curated pathway from expert analysis

flowchart TD
    A["LRRK2 G2019S<br/>Gain of Function"]
    B["RAB29 Recruitment<br/>to Lysosomal Membrane"]
    C["Enhanced Lysosomal<br/>Volume Sensing"]
    D["Signal Amplification<br/>Pathological Threshold"]
    E["Lysosomal Dysfunction<br/>Autophagy Impairment"]
    F["Neuronal Death<br/>PD Progression"]
    G["RAB29 as Modifier<br/>of LRRK2 Pathogenesis"]
    A --> B
    B --> C
    C --> D
    D --> E
    E --> F
    A --> G
    G --> C
    style A fill:#b71c1c,stroke:#ef9a9a,color:#ef9a9a
    style F fill:#b71c1c,stroke:#ef9a9a,color:#ef9a9a
    style G fill:#1b5e20,stroke:#a5d6a7,color:#a5d6a7

⚖️ Evidence

⚖️ Evidence Matrix8 supports2 contradicts
Supports
RAB29 pathogenic mutations (PARK23) cause early-onset Parkinsonism
PMID:28165311
Supports
RAB29 recruits LRRK2 to stressed lysosomes via GTP-dependent mechanism
PMID:30635564
Supports
G2019S shows selectively elevated RAB10 phosphorylation at lysosomes
PMID:33448356
Supports
Endogenous Rab29 does not impact basal or stimulated LRRK2 pathway activity.
Biochem J2020PMID:33135724medium
Supports
LRRK2 and Parkinson's disease: from genetics to targeted therapy.
Ann Clin Transl Neurol2023PMID:37021623medium
Supports
LRRK2 in Parkinson's disease: upstream regulation and therapeutic targeting.
Trends Mol Med2024PMID:39153957medium
Supports
LRRK2 mediates tubulation and vesicle sorting from lysosomes.
Sci Adv2020PMID:33177079medium
Supports
The Cell Biology of LRRK2 in Parkinson's Disease.
Mol Cell Biol2021PMID:33526455medium
Contradicts
Recruitment enhancement of G2019S relative to WT not directly demonstrated
PMID:31511666
Contradicts
Positive feedback loop mechanism not shown
PMID:30635564
📖 Linked Papers

No linked papers recorded for this hypothesis yet.

🏥 Translation

🧬 3D Protein Structure — LRRK2

🧬 PDB 6VP6 Click to expand

Experimental structure from RCSB PDB | Powered by Mol*

🧠 GTEx v10 Brain ExpressionJSON

Median TPM across 13 brain regions for LRRK2,RAB29 from GTEx v10.

Frontal Cortex BA93.5 Cortex3.3median TPM (GTEx v10)

💉 Clinical Trials

No clinical trials data linked to this hypothesis yet.

No curated ClinVar variants loaded for this hypothesis.

Run scripts/backfill_clinvar_variants.py to fetch P/LP/VUS variants.

🔍 Search ClinVar for LRRK2,RAB29 →

No DepMap CRISPR Chronos data found for LRRK2,RAB29.

Run python3 scripts/backfill_hypothesis_depmap.py to populate.

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📊 Market Indicators

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💾 Resource Usage

No resource usage or linked notebooks recorded for this hypothesis yet.

🔮 Predictions

🔎 Predictions vs Observations2 predictions · 0 with recorded observations
PredictionPredictedObservedStatusConf
IF RAB29 is knocked out via CRISPR-Cas9 in G2019S LRRK2 KI neurons, THEN the amplified RAB10 phosphorylation slope observed under nigericin-induced lysosomal swelling will be abolished, reducing the kRAB29 knockout eliminates the G2019S-specific enhancement of RAB10 phosphorylation kinetics, resulting in no significant slope difference between G2019S/RAB29-K— no observation —pending0.72
IF primary cortical neurons from G2019S LRRK2 knock-in mice are treated with nigericin (1 µM) to induce lysosomal swelling, THEN the initial rate (slope, 0-30 min) of RAB10 phosphorylation at Thr72 wiG2019S neurons show a significantly steeper rise in RAB10-pThr72 signal over the first 30 minutes of lysosomal swelling compared to WT neurons, with the slope d— no observation —pending0.78
🔮 Falsifiable Predictions (2)
pendingconf 78%
IF primary cortical neurons from G2019S LRRK2 knock-in mice are treated with nigericin (1 µM) to induce lysosomal swelling, THEN the initial rate (slope, 0-30 min) of RAB10 phosphorylation at Thr72 will increase significantly more (>50% steeper) compared to WT neurons, as measured by quantitative ph
Predicted outcome: G2019S neurons show a significantly steeper rise in RAB10-pThr72 signal over the first 30 minutes of lysosomal swelling compared to WT neurons, with t
Falsification: If the kinetic slope (rate of RAB10 phosphorylation increase) is not significantly different between G2019S and WT neurons (95% CI overlap) OR if only baseline/offset differences exist without slope d
pendingconf 72%
IF RAB29 is knocked out via CRISPR-Cas9 in G2019S LRRK2 KI neurons, THEN the amplified RAB10 phosphorylation slope observed under nigericin-induced lysosomal swelling will be abolished, reducing the kinetic difference to <15% compared to WT+RAB29-KO neurons.
Predicted outcome: RAB29 knockout eliminates the G2019S-specific enhancement of RAB10 phosphorylation kinetics, resulting in no significant slope difference between G201
Falsification: If G2019S/RAB29-KO neurons retain significantly steeper RAB10 phosphorylation slopes (>30% difference) compared to WT/RAB29-KO under lysosomal swelling, the RAB29-dependence of the amplification mecha
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