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Reactive Astrocytes A1 Phenotype in Neurodegeneration

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cell829 wordssynced 2026-04-02

Reactive Astrocytes A1 in Neurodegeneration

<table class="infobox infobox-cell">
<tr>
<th class="infobox-header" colspan="2">Reactive Astrocytes A1 Phenotype in Neurodegeneration</th>
</tr>
<tr>
<td class="label">Category</td>
<td>Examples</td>
</tr>
<tr>
<td class="label">Complement proteins</td>
<td>C3, C4</td>
</tr>
<tr>
<td class="label">Inflammatory mediators</td>
<td>IL-1β, TNF</td>
</tr>
<tr>
<td class="label">Cytokines</td>
<td>CCL2, CXCL10</td>
</tr>
<tr>
<td class="label">Stress proteins</td>
<td>[GFAP](/entities/gfap), Vimentin</td>
</tr>
</table>

Introduction

Reactive [astrocytes](/entities/astrocytes), particularly the A1 phenotype, have emerged as critical contributors to neurodegenerative disease progression. Originally characterized by Liddelow et al. in 2017, A1 astrocytes are induced by microglial release of IL-1α, TNF, and C1q, and they acquire a neurotoxic phenotype that can harm [neurons](/entities/neurons) and oligodendrocytes. Understanding A1 astrocytes is essential for developing therapeutic strategies targeting neuroinflammation in Alzheimer's disease, Parkinson's disease, ALS, and other neurodegenerative conditions. [@liddelow2017]

Overview

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