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Metabolic Pathway-Targeted Therapy in ALS

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experiment733 wordssynced 2026-04-02

Metabolic Pathway-Targeted Therapy in ALS

Experiment Overview

Rank: 109 | Score: 72/100 | Category: Translational | Disease: Amyotrophic Lateral Sclerosis

Hypothesis

Targeting systemic metabolic dysfunction — including hypermetabolism, lipid dysregulation, and glucose intolerance — will slow ALS progression and improve survival.

Knowledge Gap Addressed

Addresses the "Systemic Metabolic Dysfunction in ALS Progression" gap — specifically, whether metabolic interventions can modify disease course.

Scientific Rationale

ALS involves significant systemic metabolic dysfunction:

  • Hypermetabolism: ~60% of ALS patients have elevated resting energy expenditure
  • Lipid alterations: Reduced HDL, elevated triglycerides correlate with faster progression
  • Glucose intolerance: Insulin resistance observed in subset
  • Muscle hypometabolism: Early metabolic defects in muscle

Metabolic dysfunction may be:
  • A driver of progression (via energy deficit)
  • A consequence (via denervation)
  • Both (feedback loop)

Targeting metabolism could preserve muscle function, support neural energetics, and slow progression.

Why This Matters

  • ALS median survival is 2-4 years
  • Riluzole and edaravone provide limited benefit
  • Metabolic interventions are low-risk
  • May preserve function even if not disease-modifying

Validation Protocol

Study Design

Type: Multi-arm randomized controlled trial

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