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NETosis in Neurodegeneration

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mechanism976 wordssynced 2026-04-02

NETosis in Neurodegeneration

Related Mechanisms: [Neuroinflammation](/mechanisms/neuroinflammation), [Microglial Activation](/mechanisms/microglial-phagocytosis), [Blood-Brain Barrier Breakdown](/mechanisms/blood-brain-barrier-breakdown)

Overview

NETosis is a specialized form of cell death in neutrophils that results in the release of neutrophil extracellular traps (NETs) — webs of chromatin fibers decorated with antimicrobial proteins including neutrophil elastase (NE), myeloperoxidase (MPO), and histones[@brinkmann2004]. Originally described as a defense mechanism against pathogens, dysregulated NETosis has been increasingly implicated in the pathogenesis of neurodegenerative diseases including Alzheimer's disease (AD) and Parkinson's disease (PD)[@caielli2022].

Molecular Mechanism

Classical NETosis Pathway

Neutrophil activation triggers a cascade of events leading to NET release:

  • Neutrophil activation — via receptors (TLRs, complement receptors, Fcγ receptors) or pathogens
  • NADPH oxidase activation — produces [reactive oxygen species](/entities/reactive-oxygen-species) (ROS)
  • Peptidylarginine deiminase 4 (PAD4) activation — citrullinates histones
  • Chromatin decondensation — nuclear membrane breakdown
  • NET release — chromatin and associated proteins expelled extracellularly[@papayannopoulos2017]
  • NETosis Pathway in Neurodegeneration


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