% Bibliography for hypothesis h-var-9d9dc08d4d
% Title: GluN2B-Mediated Microglial Activation and Tau Phagocytosis
% Generated: 2026-04-28T07:35:26Z

@article{pmid35379353,
  title = {Differential vulnerability of hippocampal CA3-CA1 synapses to Aβ.},
  journal = {Acta Neuropathol Commun},
  year = {2022},
  pmid = {35379353},
}

@article{pmid31085626,
  title = {Tau accumulation triggers STAT1-dependent memory deficits by suppressing NMDA receptor expression.},
  journal = {EMBO Rep},
  year = {2019},
  pmid = {31085626},
}

@article{pmid24761931,
  title = {Chronic brain inflammation causes a reduction in GluN2A and GluN2B subunits of NMDA receptors and an increase in the phosphorylation of mitogen-activated protein kinases in the hippocampus.},
  journal = {Mol Brain},
  year = {2014},
  pmid = {24761931},
}

@article{pmid15212844,
  title = {Activation of NR1a/NR2B receptors by soluble factors from APP-stimulated monocyte-derived macrophages: implications for the pathogenesis of Alzheimer's disease.},
  journal = {Neurobiol Aging},
  year = {2004},
  pmid = {15212844},
}