Sevoflurane-induced neurotoxicity (SIN) rat model

Validation Score: 0.950 Price: $0.50 perioperative neurocognitive disorders rats Status: proposed

What This Experiment Tests

Validation experiment designed to validate causal mechanisms targeting C1qa in rats. Primary outcome: cognitive dysfunction and anxiety-like behaviors

Description

This comprehensive study established a rat model of prolonged sevoflurane anesthesia to investigate the mechanisms underlying perioperative neurocognitive disorders. The experiment involved exposing rats to prolonged sevoflurane anesthesia and then conducting extensive behavioral, molecular, and morphological analyses. Behavioral testing included Morris water maze for cognitive function, elevated plus maze and open field test for anxiety-like behaviors. The study employed multiple techniques including transcriptomic analysis (RNA sequencing), electrophysiology, molecular biology assays, scanning electron microscopy, Golgi staining, TUNEL assay, and morphological analysis to characterize the neuropathological changes. The research revealed that prolonged anesthesia triggered NF-κB inflammatory pathway activation, neuroinflammation, reduced neuronal excitability, cognitive dysfunction, and anxiety-like behaviors. RNA sequencing showed downregulation of synaptic genes, while microglial activation, migration, and phagocytosis were enhanced.

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TARGET GENE
C1qa
MODEL SYSTEM
rats
ESTIMATED COST
$0
TIMELINE
0 months
PATHWAY
complement cascade, NF-κB inflammatory pathway
SOURCE
extracted_from_pmid_36600274
PRIMARY OUTCOME
cognitive dysfunction and anxiety-like behaviors

Scoring Dimensions

Info Gain 0.00 (25%) Feasibility 0.00 (20%) Hyp Coverage 0.00 (20%) Cost Effect. 0.00 (15%) Novelty 0.00 (10%) Ethical Safety 0.00 (10%) 0.950 composite

📖 Wiki Pages

C1QA GenegeneC1QA Gene — Complement Component 1q A ChaingeneRNA Interference (RNAi) Therapies for NeurodegenertherapeuticRNA-Based Therapeutics for Neurodegenerative DiseatherapeuticRNA Targeting Therapy for NeurodegenerationtherapeuticRNA-Targeting Therapies for Neurodegenerative DisetherapeuticRNA-Based Therapeutics for Alzheimer's DiseasetherapeuticRNA Editing TherapeuticstechnologyRNA Toxicity PathwaymechanismRNA-Targeted Therapeutics Investment SynthesismechanismRNA-Targeted Therapies in NeurodegenerationmechanismRNA Stability and DecaymechanismRNA Splicing Defects in NeurodegenerationmechanismRNA Splicing Dysregulation in 4R-Tauopathies: A ComechanismRNA Splicing in Neurodegenerationmechanism

Protocol

Phase 1: Animal Preparation and Anesthesia Protocol — Week 1
Use 8-week-old male Sprague-Dawley rats (n=12 per group, Charles River) weighing 250-300g. Randomize into groups: control (room air), short sevoflurane (2h), and prolonged sevoflurane (6h). Fast animals 12h before anesthesia. Induce anesthesia with 8% sevoflurane in 100% O2, maintain at 3-4% with continuous monitoring of heart rate, respiratory rate, SpO2, and rectal temperature (maintain 37±0.5°C). Monitor blood glucose and arterial blood gases every 2h. Control group receives identical handling without anesthesia. Include positive control group with LPS injection (1 mg/kg IP, Sigma L2630) 24h before sacrifice.

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Expected Outcomes

  • 1. Primary: Prolonged sevoflurane will increase MWM escape latency by >50% and reduce probe trial time in target quadrant by >30% (p < 0.01)
  • 2. Secondary: EPM open arm time will decrease by 40-60% indicating increased anxiety-like behavior
  • 3. Tertiary: C1qa, C1qb, C1qc mRNA levels will increase 3-5 fold in sevoflurane groups compared to controls
  • 4. Quaternary: Microglial density will increase 2-3 fold with activated morphology (reduced process length and branching)
  • 5. Electrophysiological: LTP magnitude will be reduced by 35-50% in sevoflurane-exposed animals
  • 6.

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Success Criteria

  • • Behavioral significance: p < 0.05 with Cohen's d > 0.8 for MWM learning and memory measures
  • • Physiological stability: <10% variation in temperature, heart rate during anesthesia with >95% survival rate
  • • RNA quality: RIN scores >7.0 for all RNA-seq samples with >20 million mapped reads per sample
  • • Histological quality: <5% tissue damage in processed sections with successful immunostaining in >90% of samples
  • • Electrophysiological criteria: Stable baseline recordings for ≥20 minutes with LTP lasting ≥60 minutes in controls
  • • Reproducibility: Key findings (behavioral deficits, C1q

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