gene

SLC39A7

Entity Detail — Knowledge Graph Node

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This page aggregates everything SciDEX knows about SLC39A7: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

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Summary

Page for SLC39A7 — Solute Carrier Family 39 Member 7

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🧬 Gene Info

Gene Symbol	SLC39A7
Full Name	SLC39A7 — Solute Carrier Family 39 Member 7
Aliases	Solute Carrier Family 39 Member 7
Chromosome	6p21.33
Protein Type	Gene
Function	encodes a zinc transporter protein that localizes to the endoplasmic reticulum (ER) and Golgi apparatus[@huang2018].
GeneCards	SLC39A7
Human Protein Atlas	SLC39A7
Amyotrophic Lateral Sclerosis (ALS)	ER stress is a hallmark of motor neuron disease; ZIP7 dysfunction may exacerbate protein aggregation
Huntington's Disease	Zinc homeostasis disruption may contribute to mitochondrial dysfunction
FTD (Frontotemporal Dementia)	Similar ER stress mechanisms may apply
Associated Diseases	neurodegeneration
Databases	GeneCards UniProt NCBI Gene HPA STRING

Wiki Pages (1)

Knowledge base pages for this entity

Canonical Page

SLC39A7 — Solute Carrier Family 39 Member 7

gene · 717 words

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Pathway Diagram

graph TD
    SLC39A7["SLC39A7"]
    neurodegeneration["neurodegeneration"]
    SLC39A7 -->|"implicated_in"| neurodegeneration
    style SLC39A7 fill:#4a1a6b,stroke:#4fc3f7,stroke-width:2px,color:#e0e0e0

Outgoing (0)

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Incoming (0)

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Targeting Hypotheses (0)

Hypotheses where this entity is a therapeutic target

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Mentioning Analyses (0)

Scientific analyses that reference this entity

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Experiments (0)

Experimental studies targeting or related to this entity

Experiment	Type	Disease	Score	Feasibility	Model	Status	Est. Cost
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Related Papers (0)

Scientific publications cited in analyses involving this entity

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Debates (0)

Multi-agent debates referencing this entity

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Related Research

Hypotheses and analyses mentioning SLC39A7 in their description or question text

No additional research found