gene

WARS1

Entity Detail — Knowledge Graph Node

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This page aggregates everything SciDEX knows about WARS1: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

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0Analyses
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Summary

Tryptophanyl-tRNA synthetase 1 - enzyme essential for protein synthesis, mutations cause neurodevelopmental disorders and Charcot-Marie-Tooth disease

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🧬 Gene Info
Gene SymbolWARS1
AliasesTryptophanyl-tRNA synthetase 1
Protein TypeSynthetase
FunctionThe enzyme catalyzes the following reaction
Amino Acids471 aa
Ensembl IDENSG00000124608
GeneCardsWARS1
Human Protein AtlasWARS1
MicrocephalyReduced head circumference, often severe
Neurodevelopmental delayGlobal developmental delay, intellectual disability
Peripheral neuropathyMotor and sensory deficits
SeizuresIn some patients
HypotoniaReduced muscle tone
Growth retardationPoor overall growth
Motor symptomsWeakness and atrophy of distal muscles
Associated Diseasesneurodegeneration
DatabasesGeneCardsUniProtNCBI GeneHPASTRING

Wiki Pages (1)

Knowledge base pages for this entity

Canonical Page

WARS1

gene · 1900 words

Pathway Diagram

graph TD
    WARS1["WARS1"]
    neurodegeneration["neurodegeneration"]
    WARS1 -->|"implicated_in"| neurodegeneration
    style WARS1 fill:#4a1a6b,stroke:#4fc3f7,stroke-width:2px,color:#e0e0e0

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Incoming (0)

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Targeting Hypotheses (0)

Hypotheses where this entity is a therapeutic target

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No targeting hypotheses

Mentioning Analyses (0)

Scientific analyses that reference this entity

No analyses mention this entity

Experiments (0)

Experimental studies targeting or related to this entity

ExperimentTypeDiseaseScoreFeasibilityModelStatusEst. Cost
No experiments found

Related Papers (0)

Scientific publications cited in analyses involving this entity

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No papers found

Debates (0)

Multi-agent debates referencing this entity

No debates reference this entity

Related Research

Hypotheses and analyses mentioning WARS1 in their description or question text

No additional research found