disease 3,097 words KG: ent-dise-090d4eaa
Contents

ataxia-telangiectasia

Disease Info
N-terminal domainContains the FAT (FRAP/ATM/TRRAP) domain involved in protein-protein interactions
Kinase domainThe catalytic domain at positions 2719-2960 contains the conserved PI3KK active site
C-terminal regionContains the PRD (PIKK regulatory domain) and FATC (FAT C-terminal) domains essential for kinase activity
Leucine zipper regionsTwo regions (positions 760-850 and 1230-1300) that may mediate dimerization
p53Phosphorylation at Ser15 promotes p53 stabilization and transcriptional activation of DNA repair genes
Chk2Phosphorylation at Thr68 leads to cell cycle arrest
H2AXPhosphorylation at Ser139 (γH2AX) creates foci that amplify the DNA damage signal
BRCA1Phosphorylation facilitates homologous recombination repair
NF-κBActivation leads to expression of anti-apoptotic genes
IgA deficiencyPresent in 70-80% of patients, often with IgE elevation
IgG subclass deficiencyParticularly IgG2 and IgG4
Cell-mediated defectsReduced T-cell counts and function
DatabasesOMIMOrphanetClinicalTrialsPubMed

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