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Fig. 4 — Relationship between cerebral small vessel disease and proteinopathies in the me
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Fig. 4Figure 4
Association between arteriolosclerosis and perivascular spaces (PVS). Example of a section of the hippocampal body/posterior parahippocampal cortex ( A ) and of the amygdala/rhinal cortex ( B ), both stained for luxol fast blue with Hematoxylin&Eosin (LHE). PVS were present in the hippocampus and in white matter of the parahippocampal gyrus, as well as in the white matter adjacent to the rhinal cortex. PVS often displayed a vessel with severe arteriolosclerosis in the middle (insets in A and B). Big spaces at the folding sites of the hippocampus, which could potentially represent hippocampal cysts (example in A, arrow) were manually excluded from the analysis. Arteriolosclerosis severity had a main positive effect on the percentage area of the PVS, in a linear mixed effect model that included arteriolosclerosis, age at death, and sex as fixed-factors and Braak-stage group, case, and region of interest (ROI) as random-factors ( C ). The interaction between arteriolosclerosis and cerebra
▸Metadata
| pmid | 55098313-7105-44ac-8daf-b540f61c267e |
| caption | Association between arteriolosclerosis and perivascular spaces (PVS). Example of a section of the hippocampal body/posterior parahippocampal cortex ( A ) and of the amygdala/rhinal cortex ( B ), both |
| image_url | https://www.ebi.ac.uk/europepmc/articles/PMC12265290/bin/40478_2025_2076_Fig4_HTML.jpg |
| paper_title | Relationship between cerebral small vessel disease and proteinopathies in the medial temporal lobe. |
| figure_label | Fig. 4 |
| figure_number | 4 |
| _schema_version | 1 |
| source_strategy | pmc_api |
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