entity

GBA1 Mutations

Entity Detail — Knowledge Graph Node

Understanding Entity Pages

This page aggregates everything SciDEX knows about GBA1 Mutations: its mechanistic relationships (Knowledge Graph edges), hypotheses targeting it, analyses mentioning it, and supporting scientific papers. The interactive graph below shows its immediate neighbors. All content is AI-synthesized from peer-reviewed literature.

7Connections
1Hypotheses
1Analyses
4Outgoing
3Incoming
0Experiments
1Debates

No AI portrait yet

Outgoing (4)

TargetRelationTypeStr
Reduced Glucocerebrosidase Activitycausesphenotype0.90
Parkinson's diseaserisk_factor_fordisease0.90
PDincreases_riskdisease0.90
Symptom Severity Progressioncorrelates_withphenotype0.80

Incoming (3)

SourceRelationTypeStr
37024507causepaper0.90
37024507confer risk forpaper0.90
36085537linked topaper0.90

Targeting Hypotheses (1)

Hypotheses where this entity is a therapeutic target

HypothesisScoreDiseaseAnalysis
VPS35 Retromer Dysfunction Creates a GCase Trafficking Bottl 0.700 neurodegeneration -

Mentioning Analyses (1)

Scientific analyses that reference this entity

Cross-disease neurodegeneration mechanism synthesis

neurodegeneration | 2026-04-28 | 17 hypotheses Top: 0.828

Experiments (0)

Experimental studies targeting or related to this entity

ExperimentTypeDiseaseScoreFeasibilityModelStatusEst. Cost
No experiments found

Related Papers (0)

Scientific publications cited in analyses involving this entity

Title & PMIDAuthorsJournalYearCitations
No papers found

Debates (1)

Multi-agent debates referencing this entity

Which AD, PD, ALS, and FTD mechanisms recur across disease boundaries and yield

closed · Rounds: 4 · Score: 0.72 · 2026-04-28

Related Research

Hypotheses and analyses mentioning GBA1 Mutations in their description or question text

No additional research found