Network pharmacology analysis of QTJD active compounds and pathways

Exploratory Score: 0.900 Price: $0.50 Mycoplasma pneumoniae pneumonia computational network analysis Status: proposed

What This Experiment Tests

Exploratory experiment designed to discover new patterns targeting JUN, TNF in computational network analysis. Primary outcome: Identification of active compounds and therapeutic targets

Description

Network pharmacology approach was used to identify active compounds in Qingfei Tongluo Jiedu formula (QTJD) and their potential mechanisms in treating Mycoplasma pneumoniae pneumonia. High-performance liquid chromatography-electrospray ionization mass spectrometry (HPLC-ESI-MS) was combined with network pharmacology to identify 20 active compounds. Protein-protein interaction analysis revealed 10 core targets including JUN and TNF. Enrichment analysis highlighted key pathways including MAPK and PI3K-AKT signaling pathways involved in the therapeutic effects of QTJD against MPP.

TARGET GENE
JUN, TNF
MODEL SYSTEM
computational network analysis
ESTIMATED COST
$0
TIMELINE
0 months
PATHWAY
MAPK signaling pathway, PI3K-AKT signaling pathway
SOURCE
extracted_from_pmid_41754753
PRIMARY OUTCOME
Identification of active compounds and therapeutic targets

Scoring Dimensions

Info Gain 0.00 (25%) Feasibility 0.00 (20%) Hyp Coverage 0.00 (20%) Cost Effect. 0.00 (15%) Novelty 0.00 (10%) Ethical Safety 0.00 (10%) 0.900 composite

📖 Wiki Pages

TNF Alpha ProteinproteinPI3K/Akt Activator Clinical Trials for Parkinson'sclinicalTNF (Redirect)redirectJUN GenegenePI3K — Phosphoinositide 3-KinasegeneTNF - Tumor Necrosis FactorgeneTNF GenegeneMAPK Signaling Inhibitors: Investment Landscape AninvestmentAKT (Protein Kinase B) NeuronscellMAPK/ERK Signaling Pathway in NeurodegenerationmechanismMAPK/ERK Signaling Pathway in NeurodegenerationmechanismMAPK/ERK and JAK-STAT Signaling in 4R-TauopathiesmechanismMAPK Signaling Pathways in NeurodegenerationmechanismPI3K/AKT/mTOR Signaling Pathway in NeurodegeneratimechanismPI3K/Akt Signaling in Neurodegenerationmechanism

Protocol

HPLC-ESI-MS combined with network pharmacology analysis, protein-protein interaction network construction, pathway enrichment analysis

Expected Outcomes

Identification of key active compounds and signaling pathways mediating QTJD therapeutic effects

Success Criteria

Identification of biologically relevant compounds and pathways

Related Hypotheses (2)

TNF-α/IL-1β-Cx43 Hemichannel Axis as Upstream Link Between SASP and Synaptic Pruning0.663
Astrocyte-Microglia Communication Rebalancing via Cytokine Modulation0.655

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