Cerebrovascular function analysis in CD2AP mutant mice

Validation Score: 0.900 Price: $0.50 Alzheimer's disease mice Status: proposed

What This Experiment Tests

Validation experiment designed to validate causal mechanisms targeting CD2AP in mice. Primary outcome: cerebrovascular function parameters

Description

Comprehensive analysis of cerebrovascular function in mice with reduced brain endothelial CD2AP, including blood flow regulation at rest and during neurovascular coupling. The study examined mural cell activity and vascular responses, revealing altered blood flow regulation and defects in mural cell function. Sex-dependent differences were observed in vascular responses to amyloid-β treatment.

TARGET GENE
CD2AP
MODEL SYSTEM
mice
ESTIMATED COST
$0
TIMELINE
0 months
PATHWAY
neurovascular coupling, blood flow regulation
SOURCE
extracted_from_pmid_39892386
PRIMARY OUTCOME
cerebrovascular function parameters

Scoring Dimensions

Info Gain 0.00 (25%) Feasibility 0.00 (20%) Hyp Coverage 0.00 (20%) Cost Effect. 0.00 (15%) Novelty 0.00 (10%) Ethical Safety 0.00 (10%) 0.900 composite

📖 Wiki Pages

CD2AP ProteinproteinCD2AP Synaptic Dysfunction Alzheimer's Disease CaumechanismCD2AP Gene — CD2-Associated ProteingeneTREM2 Variants in Alzheimer's DiseasediseaseSporadic vs Familial Alzheimer's Disease: ComprehediseasePSEN2 Mutations in Alzheimer's DiseasediseasePSEN1 Mutations in Alzheimer's DiseasediseaseProdromal Alzheimer's DiseasediseasePreclinical Alzheimer's DiseasediseaseInvestment Landscape: Alzheimer's DiseasediseaseEarly-Onset Alzheimer's Disease (EOAD)diseaseDLB, Parkinson's Disease, and Alzheimer's Disease:diseaseAPP Mutations in Alzheimer's DiseasediseaseAlzheimer's Disease vs Parkinson's Disease ComparidiseaseAlzheimer's Disease Genetic Variantsdisease

Protocol

measurement of blood flow regulation, neurovascular coupling, mural cell activity assessment

Expected Outcomes

altered blood flow regulation and mural cell defects

Success Criteria

significant changes in cerebrovascular parameters compared to controls

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