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Gaps

3,545 items
IdTitleStatusPriority ScoreComposite ScoreCreated AtDebate CountLast Debated AtDomain
gap-pubmed-20260410-What determines whether BBB dysfunction causes or results from neuroinflammationopen0.800.002026-04-100neuroinflammation
gap-pubmed-20260410-How does Piezo1's dynamic structural complexity affect drug binding and channel open0.770.002026-04-100neurodegeneration
gap-pubmed-20260410-What are the specific mechanotransduction pathways linking Piezo1 dysfunction toopen0.790.002026-04-100neurodegeneration
gap-pubmed-20260410-How can Piezo1-targeted drugs achieve tissue-specific targeting given its widespopen0.780.002026-04-100neurodegeneration
gap-pubmed-20260410-How do platelet immune functions contribute to neuroinflammation and neurodegeneopen0.760.002026-04-100neurodegeneration
gap-pubmed-20260410-What are the specific molecular mechanisms by which platelets interact with antiopen0.770.002026-04-100neuroinflammation
gap-pubmed-20260410-Why does elevated hippocampal soluble Aβ from meprin β not produce detectable beopen0.760.002026-04-100neurodegeneration
gap-pubmed-20260410-What mechanisms drive the hippocampal vulnerability to meprin β-mediated Aβ elevopen0.800.002026-04-100neurodegeneration
gap-pubmed-20260410-Can therapeutic interventions restore oligodendrogenesis capacity in aged brainsopen0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-Why do Olig2+/Nestin+ cells show reduced proliferation while Olig2+/PDGFRα+ cellopen0.750.002026-04-100neurodegeneration
gap-pubmed-20260410-What molecular mechanisms underlie the age-related decline in OPC proliferation open0.800.002026-04-100neurodegeneration
gap-pubmed-20260410-What determines whether environmental factors cause tauopathy versus other neuroopen0.700.002026-04-100neurodegeneration
gap-pubmed-20260410-Why do most disease-modifying therapies targeting tau and Aβ pathology consistenresolved0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-What mechanisms determine tau isoform specificity and cellular targeting in diffopen0.800.002026-04-100neurodegeneration
gap-pubmed-20260410-Why has quercetin's potential in GBM treatment been inadequately explored despitopen0.790.002026-04-100neuro-oncology
gap-pubmed-20260410-What are the specific mechanisms by which quercetin overcomes drug resistance inopen0.800.002026-04-100neuro-oncology
gap-pubmed-20260410-Why does CRY1 show opposite associations with myelin integrity compared to otheropen0.770.002026-04-100neurodegeneration
gap-pubmed-20260410-How does glymphatic dysfunction causally mediate the relationship between poor sopen0.790.002026-04-100neurodegeneration
gap-pubmed-20260410-What molecular mechanisms link circadian gene expression to regional myelin vulnresolved0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-How do structural differences in STT3A versus STT3B catalytic subunits mechanistopen0.560.002026-04-100protein-folding-and-neurodegeneration
gap-pubmed-20260410-What molecular mechanisms determine the distinct substrate affinities of OST-A vopen0.640.002026-04-100protein-folding-and-neurodegeneration
gap-pubmed-20260410-What specific molecular mechanisms govern TOM-αSyn interactions and their downstopen0.830.002026-04-100neurodegeneration
gap-pubmed-20260410-How does αSyn become pathologically accumulated in PD despite not being upregulaopen0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-What are the stepwise biological mechanisms of αSyn misimport into mitochondria open0.850.002026-04-100neurodegeneration
gap-pubmed-20260410-What determines the cell-type-specific mitochondrial phenotypes associated with open0.740.002026-04-100neurodegeneration
gap-pubmed-20260410-How does ApoE4-induced fatty acid oxidation deficiency in astrocytes mechanisticopen0.760.002026-04-100synaptic-biology
gap-pubmed-20260410-What molecular mechanisms cause ApoE4 to specifically impair fatty acid transporresolved0.800.002026-04-100neurodegeneration
gap-pubmed-20260410-How can therapeutic approaches be tailored to the unique neurobiological featureopen0.790.002026-04-100neurodegeneration
gap-pubmed-20260410-What specific biomarkers can distinguish depression-AD comorbidity from either copen0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-What are the specific causal mechanisms underlying the bidirectional relationshiopen0.850.002026-04-100neurodegeneration
gap-pubmed-20260410-How does microglial C1q production contribute to chronic neuronal loss in the thopen0.810.002026-04-100neuroinflammation
gap-pubmed-20260410-Why does C1q expression increase specifically in the corticothalamic system afteopen0.790.002026-04-100neuroinflammation
gap-pubmed-20260410-What molecular mechanisms link C1q upregulation to sleep spindle disruption and open0.800.002026-04-100neuroinflammation
gap-pubmed-20260410-What are the direct molecular targets of Geniposide that initiate BTK pathway moopen0.790.002026-04-100neuroinflammation
gap-pubmed-20260410-How does BTK, primarily known for B-cell signaling, regulate neuroinflammation iopen0.800.002026-04-100neuroinflammation
gap-pubmed-20260410-How functionally equivalent are iPSC-derived microglia to brain-resident microglresolved0.780.002026-04-100neurodegeneration
gap-pubmed-20260410-What molecular mechanisms underlie the differential ADP-Ca2+ response in microglopen0.800.002026-04-100neuroinflammation
gap-pubmed-20260410-How do retinal neurons internalize tau fibrils and what determines the efficiencopen0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-What molecular mechanisms drive NaTPP to produce fewer but longer fibrils comparopen0.750.002026-04-100neurodegeneration
gap-pubmed-20260410-Why do NaTPP-induced fibrils show initially low cytotoxicity but cause delayed sopen0.800.002026-04-100neurodegeneration
gap-pubmed-20260410-How can LRRK2 kinase inhibitors be designed to avoid safety liabilities in non-nresolved0.820.002026-04-100neurodegeneration
gap-pubmed-20260410-What are the cell type-specific mechanisms by which LRRK2 kinase activity causesresolved0.850.002026-04-100neurodegeneration
gap-pubmed-20260410-How do environmental factors and lifestyle specifically interact with epigeneticopen0.780.002026-04-100environmental-neuroscience
gap-pubmed-20260410-What are the specific mechanisms by which epigenetic alterations cause brain disopen0.850.002026-04-100neuroepigenetics
gap-pubmed-20260410-What determines the balance between pyroptosis, apoptosis, and necroptosis withiopen0.770.002026-04-100neuroinflammation
gap-pubmed-20260410-How do influenza viral proteins M2, NS1, and PB1-F2 mechanistically regulate ZBPopen0.750.002026-04-100neuroinflammation
gap-pubmed-20260410-What are the precise molecular mechanisms by which ZBP1 recruits RIPK3 and caspaopen0.800.002026-04-100neuroinflammation
gap-pubmed-20260410-Why do memory deficits from GABAergic LRP1 deletion only emerge at 32 weeks but open0.740.002026-04-100neurodegeneration
gap-pubmed-20260410-How does LRP1 in GABAergic neurons maintain excitatory/inhibitory balance to preopen0.760.002026-04-100synaptic-biology
gap-pubmed-20260410-What molecular mechanisms link LRP1 deficiency in GABAergic neurons to hippocampopen0.800.002026-04-100neurodegeneration
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