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Fig. 3 — Relationship between cerebral small vessel disease and proteinopathies in the me
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Created: 2026-04-21T18:29:40
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ID: paper-fig-55098313-7105-44ac-8daf-b540f6
Fig. 3Figure 3
Association between cerebral amyloid angiopathy (CAA) and proteinopathies. The plots visualize the association between CAA and Aβ-plaques, tau-tangles, and pTDP-43 in linear mixed effects models, that included CAA percentage area, age at death, and sex as fixed-factors and Braak-stage group, case, and region of interest as random-factors. CAA percentage area was positively associated with both Aβ plaques percentage area ( A ) and with density of tau-tangles ( B ). There was no significant association between CAA and density of phosphorylated-TAR-DNA-binding protein 43 (pTDP-43) cytoplasmatic inclusions ( C ). The models explained a large part of the variance of the dependent variable. Key: AM = amygdala; RC = rhinal cortex; Est. = estimate; HC = hippocampal body; PHC = posterior parahippocampal cortex
▸Metadata
| pmid | 55098313-7105-44ac-8daf-b540f61c267e |
| caption | Association between cerebral amyloid angiopathy (CAA) and proteinopathies. The plots visualize the association between CAA and Aβ-plaques, tau-tangles, and pTDP-43 in linear mixed effects models, that |
| image_url | https://www.ebi.ac.uk/europepmc/articles/PMC12265290/bin/40478_2025_2076_Fig3_HTML.jpg |
| paper_title | Relationship between cerebral small vessel disease and proteinopathies in the medial temporal lobe. |
| figure_label | Fig. 3 |
| figure_number | 3 |
| _schema_version | 1 |
| source_strategy | pmc_api |
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