| sess_SDA-2026-04-04- | Senescent cell clearance as neurodegeneration therapy | 0.95 | 7 | 3 | completed | 2026-04-04 | SDA-2026-04-04-gap-senescent-clearance-neuro |
| sess_analysis_sea_ad | What are the cell-type specific vulnerability mechanisms in Alzheimer's disease | 0.68 | 0 | 4 | completed | 2026-04-04 | analysis_sea_ad_001 |
| sess_SDA-2026-04-04- | SEA-AD Single-Cell Analysis: Cell-Type Vulnerability in Alzheimer's Disease | 0.88 | 5 | 4 | completed | 2026-04-04 | SDA-2026-04-04-analysis_sea_ad_001 |
| sess_SDA-2026-04-04- | Which metabolic biomarkers can distinguish therapeutic response from disease pro | 0.92 | 7 | 4 | completed | 2026-04-04 | SDA-2026-04-04-gap-debate-20260403-222618-c698b06a |
| sess_SDA-2026-04-04- | How do neurodegeneration gene expression patterns in SEA-AD differ from other po | 0.94 | 7 | 4 | completed | 2026-04-04 | SDA-2026-04-04-gap-debate-20260403-222549-20260402 |
| sess_SDA-2026-04-04- | How do astrocyte-neuron metabolic interactions change during disease progression | 0.93 | 7 | 4 | completed | 2026-04-04 | SDA-2026-04-04-gap-debate-20260403-222618-e6a431dd |
| sess_SDA-2026-04-03- | The debate mentioned gene expression profiling but did not specify which neural | 0.93 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-debate-20260403-222543-20260402 |
| sess_SDA-2026-04-03- | While ketone metabolism was discussed as therapeutic, the debate revealed no cle | 0.80 | 8 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-debate-20260403-222618-2709aad9 |
| sess_SDA-2026-04-03- | The debate highlighted TFEB's role in mitochondrial-lysosomal coupling but could | 0.95 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-debate-20260403-222617-8eb5bdbc |
| sess_SDA-2026-04-03- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.95 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-aging-mouse-brain-v3-20260402 |
| sess_SDA-2026-04-03- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.95 | 6 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-aging-mouse-brain-v2-20260402 |
| sess_SDA-2026-04-03- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.95 | 6 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-aging-mouse-brain-20260402 |
| sess_SDA-2026-04-03- | Evaluate the potential of CRISPR/Cas9 and related gene editing technologies for | 0.95 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-crispr-neurodegeneration-20260402 |
| sess_SDA-2026-04-03- | Comprehensive analysis of immune cell subtypes in neurodegeneration: microglia s | 0.85 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-immune-atlas-neuroinflam-20260402 |
| sess_SDA-2026-04-03- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.92 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-seaad-20260402025452 |
| sess_SDA-2026-04-03- | Analyze circuit-level changes in neurodegeneration using Allen Institute Neural | 0.95 | 6 | 4 | completed | 2026-04-03 | SDA-2026-04-03-26abc5e5f9f2 |
| sess_SDA-2026-04-03- | Investigate prion-like spreading of tau pathology through connected brain region | 0.95 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-tau-prop-20260402003221 |
| sess_SDA-2026-04-03- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.90 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-seaad-v4-20260402065846 |
| sess_SDA-2026-04-03- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.95 | 7 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-seaad-v3-20260402063622 |
| sess_SDA-2026-04-03- | What cell types are most vulnerable in Alzheimer's Disease based on SEA-AD trans | 0.95 | 6 | 4 | completed | 2026-04-03 | SDA-2026-04-03-gap-seaad-v2-20260402032945 |
| sess_SDA-2026-04-02- | What cell types are most vulnerable in Alzheimer's Disease based on SEA-AD trans | 0.95 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-seaad-v2-20260402032945 |
| debate-seaad-2026040 | Cell-type specific expression patterns of neurodegeneration genes in SEA-AD | 0.68 | 5 | 3 | completed | 2026-04-02 | analysis-SEAAD-20260402 |
| sess_SDA-2026-04-02- | How does metabolic reprogramming (glucose metabolism shifts, brain insulin resis | 0.73 | 3 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-v2-5d0e3052 |
| sess_SDA-2026-04-02- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.71 | 4 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-aging-mouse-brain-v5-20260402 |
| sess_SDA-2026-04-02- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.74 | 3 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-seaad-v4-20260402065846 |
| sess_SDA-2026-04-02- | Epigenetic reprogramming in aging neurons | 0.95 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-epigenetic-reprog-b685190e |
| sess_SDA-2026-04-02- | Extracellular vesicle biomarkers for early AD detection | 0.87 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-ev-ad-biomarkers |
| sess_SDA-2026-04-02- | Investigate prion-like spreading of tau pathology through connected brain region | 0.88 | 6 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-tau-prop-20260402003221 |
| sess_SDA-2026-04-02- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.57 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-seaad-v3-20260402063622 |
| sess_SDA-2026-04-02- | Senescent cell clearance as neurodegeneration therapy | 0.95 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-senescent-clearance-neuro |
| sess_SDA-2026-04-02- | Evaluate the potential of CRISPR/Cas9 and related gene editing technologies for | 0.90 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-crispr-neurodegeneration-20260402 |
| sess_SDA-2026-04-02- | The disease-associated microglia (DAM) phenotype involves TREM2 upregulation, bu | 0.89 | 7 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-001 |
| sess_SDA-2026-04-02- | Comprehensive analysis of immune cell subtypes in neurodegeneration: microglia s | 0.55 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-immune-atlas-neuroinflam-20260402 |
| sess_SDA-2026-04-02- | Analyze circuit-level changes in neurodegeneration using Allen Institute Neural | 0.42 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-26abc5e5f9f2 |
| sess_SDA-2026-04-02- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.43 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-aging-mouse-brain-v4-20260402 |
| sess_SDA-2026-04-02- | What cell types are most vulnerable in Alzheimers Disease based on SEA-AD transc | 0.56 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-seaad-20260402025452 |
| sess_SDA-2026-04-02- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.50 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-aging-mouse-brain-v3-20260402 |
| sess_SDA-2026-04-02- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.57 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-aging-mouse-brain-v2-20260402 |
| sess_SDA-2026-04-02- | What gene expression changes in the aging mouse brain predict neurodegenerative | 0.56 | 1 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-aging-mouse-brain-20260402 |
| sess_SDA-2026-04-02- | Analyze the spectrum of microglial activation states (DAM, homeostatic, inflamma | 0.49 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-microglial-subtypes-20260402004119 |
| sess_SDA-2026-04-02- | Is disrupted sleep a cause or consequence of neurodegeneration? Analyze the bidi | 0.59 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-20260402-003115 |
| sess_SDA-2026-04-02- | Is disrupted sleep a cause or consequence of neurodegeneration? Analyze the bidi | 0.46 | 0 | 4 | completed | 2026-04-02 | SDA-2026-04-02-gap-20260402-003058 |
| sess_SDA-2026-04-01- | The disease-associated microglia (DAM) phenotype involves TREM2 upregulation, bu | 0.57 | 7 | 4 | completed | 2026-04-01 | SDA-2026-04-01-gap-001 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying mechanistic role of apoe in neurodegeneration | 0.94 | 6 | 5 | completed | 2026-04-01 | SDA-2026-04-01-gap-auto-fd6b1635d9 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying tdp-43 phase separation therapeutics for als- | 0.95 | 7 | 4 | completed | 2026-04-01 | SDA-2026-04-01-gap-006 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying neuroinflammation resolution mechanisms and p | 0.95 | 7 | 5 | completed | 2026-04-01 | SDA-2026-04-01-gap-014 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying autophagy-lysosome pathway convergence across | 0.95 | 7 | 5 | completed | 2026-04-01 | SDA-2026-04-01-gap-011 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying mitochondrial transfer between neurons and gl | 0.81 | 7 | 5 | completed | 2026-04-01 | SDA-2026-04-01-gap-20260401231108 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying microglia-astrocyte crosstalk amplification l | 0.95 | 7 | 4 | completed | 2026-04-01 | SDA-2026-04-01-gap-009 |
| sess_SDA-2026-04-01- | What are the mechanisms underlying what are the mechanisms by which gut microbio | 0.89 | 7 | 6 | completed | 2026-04-01 | SDA-2026-04-01-gap-20260401-225155 |