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African swine fever virus hijacks lipolysis induced by chaperone-mediated autophagy to upregulate fatty acid β-oxidation and promote viral replication.
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ID: paper-41801037
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African swine fever virus hijacks lipolysis induced by chaperone-mediated autophagy to upregulate fatty acid β-oxidation and promote viral replication.
["Yang X", "Xiong X", "Liu H", "Wen X", "Shi X", "Ma H", "Wang R", "Cao W", "Yang F", "Ru Y"]
Abstract
Lipid metabolism plays a crucial role in cellular signal transduction, affects the structural integrity of cell membranes, and regulates energy metabolism. However, various viruses, including African swine fever virus (ASFV), usurp lipid metabolism to enhance their replication. The mechanism for the positive role of lipid metabolism in ASFV infection is unclear. Here, we present data that ASFV infection concurrently upregulates both fatty acid synthesis (FAS) and fatty acid β-oxidation (FAO) to ...
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