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Fig. 2 — A novel derivative of valepotriate inhibits the PI3K/AKT pathway and causes Noxa
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Created: 2026-04-21T18:29:40
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Fig. 2Figure 2
Amcp inhibited the proliferation of human pancreatic cancer cells. a The chemical structure of Amcp. b The time- and dose-dependent inhibitory effects of Amcp on two human pancreatic cancer cell lines, BxPC-3 and SW1990, in vitro. Cells were treated with Amcp at concentrations of 0.625, 1.25, 2.5, 5, and 10 µM for either 24, 48, or 72 h before the CCK-8 assay. c DAPI staining of the cell nucleus. Cells were treated with the indicated compounds for 24 h before fixation and stained with DAPI stain. Fluorescence was observed by using microscopy (Nikon Eclipse Ti). The magnification is ×400. Scale bar = 20 µm.
▸Metadata
| pmid | paper-8281d7c4ac82 |
| caption | Amcp inhibited the proliferation of human pancreatic cancer cells. a The chemical structure of Amcp. b The time- and dose-dependent inhibitory effects of Amcp on two human pancreatic cancer cell li |
| image_url | https://www.ebi.ac.uk/europepmc/articles/PMC7470838/bin/41401_2019_354_Fig2_HTML.jpg |
| paper_title | A novel derivative of valepotriate inhibits the PI3K/AKT pathway and causes Noxa-dependent apoptosis in human pancreatic cancer cells. |
| figure_label | Fig. 2 |
| figure_number | 2 |
| _schema_version | 1 |
| source_strategy | pmc_api |
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