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Tau Spreading Network Mapping via Spatial Transcriptomics in PSP

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wiki page Created: 2026-04-02T07:20:09 By: crosslink-migration Quality: 50% ✓ SciDEX ID: wiki-experiments-tau-spreading-network-m
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experiment670 wordssynced 2026-04-02

Hypothesis

Primary Hypothesis: 4R-tau pathology in PSP originates in specific vulnerability hotspots (subcortical nuclei, brainstem) and spreads trans-synaptically along anatomically connected neural networks, following prion-like templating mechanisms.

Secondary Hypothesis: The pattern of tau spreading correlates with clinical phenotypes (Richardson's syndrome vs. variants) and can be predicted by connectome architecture derived from structural/functional MRI.

Pathway / Mechanism Diagram

graph TD A["Tau Misfolding in Donor Neuron"] --> B["Tau Oligomer Formation"] B --> C["Secretion via Exosomes"] B --> D["Direct Synaptic Transfer"] B --> E["Tunneling Nanotubes"] C --> F["Extracellular Tau Uptake"] D --> F E --> F F --> G["Templated Misfolding in Recipient"] G --> H["Prion-like Propagation"] H --> I["Braak Stage Progression"] I --> J["Entorhinal Cortex (Stage I-II)"] J --> K["Hippocampus (Stage III-IV)"] K --> L["Neocortex (Stage V-VI)"] G --> M["Microglial Activation"] M --> N["Neuroinflammation"] N --> O["Accelerated Tau Spread"] O --> H style A fill:#5d4400,color:#e0e0e0 style L fill:#ef5350,color:#e0e0e0 style H fill:#006494,color:#e0e0e0

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📊 Evidence Profile Foundational
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75%
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